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By John L. Turner

John Turner

Strolling on a mid-December day to pick up the morning paper at the base of the driveway, I passed by the bird bath and noticed the surface had a thin layer of ice capping a few inches of water underneath, an event about as surprising as the fact gravity held me to the ground as I fetched to get the newspaper. 

Except that a thought I had not had for a very long time suddenly flashed to mind, from something I read in a middle school Earth Science textbook, a thought about a concept that is remarkably consequential — if ice wasn’t lighter than water but rather denser, life might not have ever gotten a foothold on planet Earth or if it did, it might have happened later and in a much more limited fashion geographically speaking.   

As water cools it becomes denser so water closer to a freezing temperature, say 40 degrees Fahrenheit, is denser than at 80 degree water and 80 degree water is denser than water near its boiling point. The fact the colder water is, the denser it is, is true — but only to a point. Once water falls below 39.4 degrees Fahrenheit it reverses course density wise and becomes less dense as water molecules shift to form a lattice-like structure of spread out interconnected hexagons (six sided) once the water freezes; this lower density explains why ice is always on the surface and why ice cubes and icebergs float.    

If water lacked this chemical quirkiness (in one scientific account characterized as ‘anomalous physical behavior’) and ice was denser than water, when ice formed at the surface from contact with air below 32 degrees, it would sink to the bottom, soon freezing solid the entire water column from bottom to top and everything in between if it stayed cold long enough. 

All turtles, frogs, salamanders, and fish would be frozen along with the much smaller zooplankton that forms the base of the aquatic food chain located in temperate climates. Aquatic mammals such as beavers, muskrats, and otters would struggle mightily to survive. The same would be true for bays, harbors and the shallow portions of oceans, creating profound difficulties for the animals living in the colder portions of the marine realm. 

If ice was denser than water would life ever have evolved on Earth? If so, would it be in the countless forms we see today? Would there have been other evolutionary pathways than the traditionally understood fish to amphibian to reptile to bird and mammal route we have deciphered from genetic evidence and the fossil record? Would you or I  even exist to read and write this article, respectively? 

Fortunately, our world is one in which water behaves oddly, with ice always floating on water, forming a protective layer for the free swimming aquatic life beneath. No matter how cold and bone-chilling the temperature of the air, even in circumstances involving temperatures much below zero (as routinely happens in mountainous areas and the polar regions), the water beneath the ice remains a ‘balmy’ 33 degrees or slightly higher, allowing for life to persist. 

And ice isn’t the only form of frozen water that protects life. Snow does the same.  One foot of snow is enough to keep the soil near 32 degrees despite what the air temperature is above the snow. This insulative value is not surprising given the fact that about 90% of the volume of a freshly fallen blanket of snow is air! 

This allows for small mammals like voles and mice to remain active through the winter, hidden from predators beneath the snow, although a life free from predation is never a guarantee; a fact borne out on a few occasions when I’ve seen both red fox and coyote spring high into the air, arching their backs to gain momentum and focus, coming hard down on the snow with their front paws to punch through the crusty surface layer of snow in pursuit of a vole or mouse it heard below. 

I well remember watching a coyote in a wind-blown, snow-covered farm field in Ontario, north of Ottawa, about thirty winters ago repeatedly pouncing through the snow, eventually catching what looked like to be a meadow vole. 

Snow also enables animals hibernating beneath (called the subnivean zone) to use less energy and worry less about frostbite during this vulnerable time. Snow also protects plants from “frostbite” by preventing the soil from freezing and damaging small roots and rootlets. That’s why snow is referred to as the “poor man’s mulch”! The snow prevents a freeze-thaw-freeze-thaw  cycle which can  push or heave a plant from the soil causing root damage.

And a snow cover benefits human animals and their properties too, by safeguarding underground water lines from freezing since slightly below the snow-covered surface the temperature remains above the freezing point. This might not be true if very cold air can make prolonged contact against a ground that lacks the benefit of a snow blanket.     

Under certain conditions though, snow and ice can prove lethal to plants and animals. The weight of wet snow can break branches and occasionally break or topple trees, especially evergreens whose abundant needled leaves hold snow. Ice forming inside plant and animal cells can be lethal as microscopically small ice shards puncture cell walls. 

The wood frog, a native amphibian that breeds in vernal pools throughout Long Island (vernal pools will be the subject of a Nature Matters column in the Spring of 2025) actually freezes solid in the winter and is able to survive by pumping water out of its cells so they stay protected.  No wonder they are amusingly called ‘frogcicles’! 

If you want to see a wood frog thawing out after a long winter of being frozen but somehow still staying alive, I invite you to look at YouTube videos. 

So there you have it — ice and snow — two substances which can disrupt life in specific situations but lifegiving in a general sense. And since we’re still in the glow of the holiday season, let’s be forever thankful for the unique, life-permitting nature of water molecules. 

A resident of Setauket, author John L. Turner is a naturalist, conservation co-chair of the Four Harbors Audubon Society, and Conservation Policy Advocate for the Seatuck Environmental Association.

These composite brain images from the study of WTC responders reveal evidence of amyloidosis. Areas shaded in red and yellow indicate regions of the brain with amyloid. Image courtesy of Sean Clouston, Stony Brook Medicine

By Daniel Dunaief

Even over 23 years after first responders raced to the smoldering site of the World Trade Center terrorist attacks, many emergency crews continue to battle the effects of their exposure.

With a combination of toxic aerosolized particles infusing the air, first responders who didn’t wear personal protective equipment and who had the highest degree of exposure have suffered from a range of symptoms and conditions.

Sean Clouston

In a recent study of 35 World Trade Center first responders in the Journal of Alzheimer’s Disease, lead author Sean Clouston, who is a Professor in the Department of Family, Population and Preventive Medicine in the Renaissance School of Medicine, found evidence of amyloid plaques, which are often linked to Alzheimer’s Disease.

The paper links exposure to a neurodegenerative protein.

Research with World Trade Center first responders not only benefits those who worked tirelessly to try to find survivors and to restore the area after the attack, but also could help other people who inhale aerosolized toxins.

Indeed, such research could help those who are spending hours battling the ongoing wildfires in Los Angeles, which have been consuming forests and trees, homes and commercial buildings, at a furious and uncontrolled pace.

People have a feeling that fresh air is safe, but what scientists have learned from their studies of the World Trade Center first responders is that “just being six feet away from a pile of rubble that’s smoldering, even if you can’t see that it’s dangerous, doesn’t mean it isn’t,” said Clouston. “There is at least some risk” to human health from fires that spew smoke from burned computers and refrigerators, among others.

Given the variety of materials burned in the fires, Minos Kritikos, Senior Research Scientist and a member of the group in the collaborative labs of Clouston and Professor Benjamin Luft, suspects that a heterogeneity of particles were in the air.

People in Los Angeles who are inhaling these particles can have them “linger in their circulation for years,” said Kritikos. “It’s not just a neurological issue” as the body tries to deal with carrying around this “noxious” particulate matter. Since most neurons don’t regenerate, any toxicity induced neuronal death is irreversible, making damage to the brain permanent.

Even in non-emergency situations, people in polluted cities face increased health risks.

“There is a recognition that air pollution is a major preventable cause of Alzheimer’s Disease and related dementias, as noted by the latest Lancet Commission,” Clouston explained.

Two likely entry points

People who breathe in air containing toxic chemicals have two likely pathways through which the particulates enter the body. They can come in through the nose and, potentially, travel directly into the brain, or they can enter the lungs, circulate through the body and enter the head through the blood-brain barrier. The olfactory route is more direct, said Kritikos. 

Minos Kritikos in front of Stony Brook Hospital.

The amyloid plaques in these first responders was found primarily in the area near the nose, which supports the idea that maybe inhaling the dust was the problem, Clouston said.

Once these chemicals enter the brain, Clouston and his team believe the body engages defenses that are designed much more for viruses than for toxic compounds. The immune system can encapsulate these chemicals in amyloid plaques.

Amyloid plaques, in moderation and under conditions that protect the brain against pathogens, are a part of a protective and helpful immune response. Too much of a good thing, however, can overwhelm the brain.

“When there’s too much plaque, it can physically disturb neuronal functions and connections,” said Kritikos. “By being a big presence, they can also molecularly and chemically react with its environment.”

A large presence of amyloid can be toxically necrotic to surrounding neural tissue, Kritikos added.

What the scientists believe they are tracking is the footprint of an adaptive response that may not help the brain, Clouston added.

Clouston cautioned that the plaques and cognitive decline could both be caused by something else that scientists haven’t yet seen.

The findings

The research, which used positron emission tomography and magnetic resonance imaging scans to search for evidence of amyloid plaques, found evidence that doesn’t look like old age Alzheimer’s, explained Clouston. Usually these levels of plaques are not located in one spot, but occur throughout the brain during Alzheimer’s. 

The immune response may be causing some of these plaques.

The amount of amyloid plaque doesn’t look like Alzheimer’s Disease and does not appear abnormal in the traditional way of testing, but with careful analysis of the olfactory system, the researchers can find elevated levels.

“I was surprised by how little amyloid was necessary to show this association,” said Clouston.

Researchers at Mt. Sinai have examined the effect of exposure to these same particulates in mice.

“The answer is very much similar to what we see in humans,” said Clouston. “That supports this work.”

To be sure, Clouston and Kritikos are hoping to build on this research. They are particularly interested in following up with participants to measure the rate of change in these plaques from the observed amyloid signals they measured at baseline.

“Doing so would enable us to calculate the rate of amyloid buildup allowing us to assess our responders more precisely, opening doors to possible therapeutic interventions such as the recently approved anti-amyloid therapies,” Kritikos explained. 

Additionally, they hope to expand on the study beyond the 35 people who participated.

It is unclear whether tamping down the immune system could make patients better or worse. By reducing amyloid plaques, scientists might enable the harmful dust to cause damage in other areas of the brain. Alternatively, however, a lower level immune response with fewer plaques might, in the longer term, be better for the brain.

This study “does open the door for some of those questions,” Clouston said. Kritikos and Clouston plan to study the presence of tau proteins and any signs of neurodegeneration in the brains of these first responders.

“More research needs to be done,” Clouston said, which specifically targets different ways of measuring exposure, such as through a biomarker. He’s hoping such a biomarker might be found that tracks levels of exposure.

Future research could also address whether post traumatic stress disorder affects the immune response.

“It’s certainly possible that PTSD is playing a role, but we’re not sure what that might be,” said Clouston.

The researchers are continuing this research as they study the effects of exposure on tau proteins and neurodegeneration.

“We are hopeful that this will be an important turning point for us,” Clouston explained

From the Medditerranean to the Atlantic

Born and raised in Cyprus, Kritikos comes from a large family who are passionate about spending time with each other while eating good food.

He earned his doctorate from the University of Bristol in England.

Kritikos met his wife Jennifer LoPresti Kritikos, who is originally from Shirley, New York, at a coffee shop in Aberdeen, Scotland, where he was doing postdoctoral research.

LoPresti, who works at Stony Brook as the Department Head Administrator for Biomedical Engineering, and Kritikos live in Manorville and have an eight year-old daughter Gia and one-year old son Theseus.

As for his work, Kritikos is grateful for the opportunity to contribute to research with Clouston and Luft, who is the Director of the Stony Brook WTC Health and Wellness Program.

“I’m happy to be in a position whereby our large WTC team (the size of a small village) is constantly pushing forward with our understanding for how these exposures have affected” the brain health of WTC first responders, Kritikos explained. He would like to continue to uncover mechanisms that underly these phenomena, not just for WTC responders but also for similarly exposed populations.

 

From left, Frankenstein and Catapotumous

Adopt Frankenstein and Catapotumous!                                                                             

This week’s shelter pets are Frankenstein and Catapotumous – Frankie and Cat for short. This inseparable duo arrived at the shelter after their owner, sadly, could no longer care for them. Their deep bond is truly heartwarming, and our greatest wish is to find them a loving home where they can enjoy their golden years together.

Frankenstein is a stunning gray and white domestic shorthair, estimated to be around 17 years young. The senior of the pair, exudes warmth and affection, winning over everyone she meets. Frankie would love nothing more than to be adopted alongside her best friend, Cat. She would do well in a home with older children, other cats, and calm dogs.

Catapotumous, is a gorgeous thirteen -year-old, brown and white, domestic short hair, female tabby.  Her affectionate nature makes her the perfect companion, ready to share her love and playfulness with a loving family. Expect a double dose of purring, lap naps and snuggles while in the company of Cat and her bestie Frankie. Cat would thrive in most home environments, including those with older kids, other cats, and calm dogs.

These two lovely ladies bring double the love and companionship—if you’re looking to add warmth and joy to your home, consider opening your heart to Frankie and Cat. They’re ready to shower their new family with endless love!

If you are interested in meeting  Frankenstein and Catapotumous please fill out an application to schedule time to properly interact with your prospective soul mate in a domestic setting.

All of the felines at the Shelter are current on vaccines and have received a full workup (blood work, Feline HIV & Leukemia tested, physical exam etc) by a board certified Veterinarian.

For more information regarding our rescue animals available for adoption visit:. TownofSmithtownAnimalShelter.com 

The Town of Smithtown Animal & Adoption Shelter is located at 410 Middle Country Road, Smithtown. Visitor hours are Monday to Saturday from 10 a.m. to 3 p.m. (Sundays and Wednesday evenings by appointment only).  For more information, call 631-360-7575 or visit www.townofsmithtownanimalshelter.com.

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By Fr. Francis Pizzarelli

Fr. Francis Pizzarelli

There is so much conflict and polarization in the world today. We need to work harder at building bridges instead of walls. DEI — diversity, equity, and inclusion — is such a powerful way to build those bridges that are so crucial.

To blame tragedies on DEI is unconscionable. DEI should be woven in all of our workplaces, in our schools and in our sports. We are a nation founded on diversity; hopefully a nation that practices equity and is continuously looking for ways to be more inclusive.

Unfortunately, discrimination, lack of equity and inclusiveness have been part of our history. Many have worked to correct those areas that are not inclusive at all and open to diversity.

Wherever DEI is present, it strengthens the workplace. It does not weaken it. Standards are not compromised. Expectations are not minimized. Inclusivity is an attempt to make sure that everyone is welcomed;  equity is an attempt to make sure everyone is treated fairly without compromise or discrimination.

To eliminate DEI would be a step backwards; it would once again reinforce prejudicial attitudes and behaviors. No matter what our political affiliation, we need to be advocates for inclusivity equity and diversity. We have made such great strides in combatting, exclusivity, injustice, and blatant discrimination. We have an opportunity as a country to transcend political differences and find a way to work together to make all Americans feel welcomed, respected, and included.

These are definitely challenging times, but also an opportunity to come together despite our differences. It is an opportunity to move forward as a stronger nation respecting the gifts and talents of all people and using their gifts and talents to build a stronger and more resilient nation.

DEI and its initiatives are aimed at fostering a culture that embraces and values differences; ultimately leading not only to better outcomes for organizations that embrace these initiatives, but also an opportunity to strengthen not only our communities, but our nation. It is an opportunity to really build strong bridges, and not walls.

DEI is a framework that can promote a more inclusive and fair environment for all individuals regardless of their backgrounds or their identities. Our nation was founded on freedom and respect for all people despite our ethnic differences. We are a melting pot nation and stronger because of that.

As we welcome a new leadership team, they represent us and what we believe is important for our country. Unity, inclusiveness, respect and equity are critical for our future and for the strength of our nation. We don’t want the next generation to be blinded by race, religion, ethnicity, or sexual orientation. We want the next generation to celebrate our diversity as a gift and a blessing.

Father Francis Pizzarelli, SMM, LCSW-R, ACSW, DCSW, is the director of Hope House Ministries in Port Jefferson.

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By Daniel Dunaief

Daniel Dunaief

With temperatures soaring, the large gathering of friends and family used programs to fan themselves.

Sweat dripped down my back as I waited in a wooden chair amid bright sunshine to witness and celebrate my brother’s graduation from Colgate University. Thoughts of my brother mixed with a need to find shade and an ice cold drink.

I had attended several graduations before this one, including my older brother’s and my own from college. I recall my Ward Melville High School graduation being incredibly warm as well, but this one seemed longer and more protracted, perhaps because we had to drive several hours to attend.

I pondered all the phone calls to the 315 area code with my younger brother, who complained about the snow, the ice and the bitter cold temperatures, which was an enormous contrast to the stifling heat of that day.

The commencement speaker, Fay Vincent, was the commissioner of baseball at the time. His tenure started when his predecessor Bart Giamatti died of a heart attack at 51, eight days after banning Pete Rose from baseball for life for gambling on the game. 

I thought about Vincent this week when I learned he had died at the age of 86 from cancer. I remember a surprising number of thoughts and ideas he shared during that momentous and proud day in our family’s life.

Fortunately, the sound system on that field was clear enough that I could hear almost every word he said.

He started off by saying that there’s no such thing as a good, long commencement speech. Similarly, there’s no such thing as a bad, short commencement speech.

The overheated audience laughed at both well-delivered lines, relieved that their children’s names would be called fairly soon.

He had three pieces of advice for these graduates. He recommended that they do something good. While that course of action depended on each person’s definition of “good,” he urged people to use their time effectively and to contribute something to society.

He then suggested that people do something well. Merely taking a job, moving on to graduate school, or doing some kind of internship wasn’t enough: graduates needed to commit to completing any task well, whatever that might be.

And, finally, he urged them to do something. That line also elicited considerable laughter from the appreciative crowd, who, by now, was eating out of his hand and wasn’t so sure they wanted this speech to end too rapidly.

He urged graduates not to return home and spend too much time pondering their future. They needed to go out and experience life. And, of course, once they took whatever next steps, they should remember to engage in good work while committing to doing it well.

Recognizing that the commissioner of baseball couldn’t get off the dais without sharing a baseball story, he also offered one about a rookie umpire.

The Houston Astros were playing the Yankees at the Astrodome in front of more than 50,000 adoring fans during their home opener.

Nolan Ryan, the ace of the team and one of the best pitchers in baseball who still holds the record with seven no hitters, was on the mound.

Mickey Rivers, the speedy Yankees center fielder, was stepping up to the plate. The umpire signaled for Ryan, who entered the Hall of Fame in 1999 as a Texas Ranger, to throw the first pitch.

The ball exploded into the catcher’s mitt. The fans waited as the seconds ticked by. The players looked in for the call. The umpire signaled weakly, “strike?” and extended his arm. The crowd cheered wildly as the catcher tossed the ball back to Ryan, who had started out his 27-year career as a member of the New York Mets.

Rivers took a step out of the box and smiled at the umpire. “Hey, it’s okay, blue,” Rivers said. “I didn’t see it either.”

Vincent told the story and offered his advice with such gusto and passion that I can still recall the speech and its effect on people all these years later. 

Is it possible that Ryan was on another team during that speech and that another batter shared such relatable words to the umpire? Sure. But, I can remember the message, the charm and the encouragement (and, of course, the searing heat) as if I were sitting in that field yesterday, celebrating my brother’s graduation and appreciating Vincent’s word’s of wisdom.

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By Leah S. Dunaief 

Leah Dunaief,
Publisher

Cancer. Half a century ago, it was a word only whispered, so dreaded was the disease. It was considered a death sentence. People who had it were often shunned, as if it were contagious. And hospital treatments were demonic. As one cancer specialist at Dana-Farber Cancer Institute in Boston put it, “We were kind of just pushing poisons and hoping for the best.”

But cancer deaths have plummeted in the last 30 years, as scientists and physicians have greatly improved their understanding of the pathology, and treatments now target some cancer-causing genes. Likened to diabetes, cancer might be thought of as a manageable chronic disease.

What exactly is cancer?

An excellent article in this Tuesday’s issue of The New York Times science section tackled that question. “Every day, billions of cells in our body divide or die off. Once in a while, though, something goes awry, and cells that should stop growing or die simply don’t. Left unchecked, those cells can turn into cancer,” writes Nina Agrawal.

Why that happens and how to treat the problem still puzzles scientists and doctors, but there has been great progress in understanding the disease in its various forms. While they used to think that mutations of genes caused all cancer, that has turned out to be only part of the story. Some mutations lay dormant an entire lifetime and never lead to cancer.

Separate from DNA code mutations, there are epigenetic changes, changes due to our environment in the way genes are expressed, that play a huge role. These may be caused by aging, dietary and environmental exposures, and chronic inflammation.

Some chemicals have long been known to cause cancer, like asbestos and those in cigarette smoke. Air pollution is now thought to increase risk, especially for lung and breast cancers, triggering inflammation. So does eating an unhealthy diet, which “can upset the balance of our microbiome, allowing certain bacteria to grow unchecked. Scientists think this may cause chronic inflammation, which can lead to colon or pancreatic cancers,”  Dr. Davendra Sohal, a gastrointestinal oncologist, told The New York Times.

Interestingly, malignant tumors are made up of cancer cells, as well as normal cells “that have been recruited to support their growth,” according to The NYT. “Many of these normal cells are the same type of immune cells that will flood the site of an injury or infection to help heal that wound—by helping new cells multiply, generating blood vessels, stimulating new connective tissue and avoiding attacks from other parts of the immune system. These are capabilities that cancer cells can co-opt indefinitely to support their own growth.”

Epidemiologists estimate that 40 percent of cancers and cancer deaths can be caused by controllable risk factors. These include cigarette smoking, sun exposure, alcohol use and excess body weight. Some infections, for example, caused by hepatitis B and C viruses, human papillomavirus and H. pylori bacteria, can also cause certain cancers, according to the article.

Understanding better how the immune system works has opened up a new treatment field called immunotherapy, using T-cells, immune system fighters produced or processed by the thymus gland that kill cancer cells in lungs and skin, among others. Engineered by doctors, T-cells, forming CAR T-cell therapy, have been most effective against blood cancers, the reporter said.

Can cancer be cured?

While physicians are reluctant to use that term, newer treatments like stem cell transplants and CAR T therapy make for optimism, especially after a number of years elapse when a patient is in remission.

Research further to develop prevention and treatments must continue.

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Lifestyle modifications including diet can help

By David Dunaief, M.D.

Dr. David Dunaief

In my practice, many patients have resisted telling me they suffered from erectile dysfunction (ED). However, it’s a common problem. Because it can indicate other medical issues, it’s important that you share this information with your doctor.

ED affects about 24 percent of men, on average. If it occurs less than 20 percent of the time, it is considered normal; however, if it occurs more than 50 percent of the time, you should seek help (1). 

Of course, there are oral medications for ED. You’ve probably seen the ads for approved medications, including sildenafil (Viagra, or the “little blue pill”), tadalafil (Cialis), vardenafil (Levitra, Staxyn), and avanafil (Stendra). They work by causing vasodilation, or enlargement of blood vessels, which increases blood flow to the penis. Unfortunately, this does not solve the medical problem, but it does provide a short-term solution for those who are good treatment candidates.

ED’s prevalence generally increases with age. An analysis of the 2021 National Survey of Sexual Wellbeing found that ED affected 12.7 percent of 35-44-year-olds, increased to 25.3 percent of 45-54- year-olds, 33.9 percent of those aged 55-64, 48 percent of those aged 65-74, and 52.2 percent of those aged 75 and older (2).

So, what contributes to the increase as we age? Disease processes and drug therapies.

What is the connection between medical conditions and ED?

Chronic diseases can contribute significantly to ED. ED might also be an indicator of disease. Typical contributors include metabolic syndrome, diabetes, high blood pressure, cardiovascular disease and obesity. In the Look AHEAD trial, ED had a greater than two-fold association with hypertension and a three-fold association with metabolic syndrome (3). In another study, ED was associated with a 2.5-times increase in cardiovascular disease (4).

Patients with ED had significantly more calcification, or atherosclerosis, in their arteries when compared to a control group in a randomized clinical trial (RCT) (5). They were also more than three times as likely to have severe calcification. In addition, they had more inflammation, measured by C-reactive protein. 

Which medications contribute to ED?

About 25 percent of ED cases are thought to be associated with medications, such antidepressants, NSAIDs (e.g., ibuprofen and naproxen sodium), and hypertension medications. Unfortunately, the most common antidepressant medications, SSRIs, have significant impacts on ED. 

The California Men’s Health Study, with over 80,000 participants, showed that there was an association between NSAIDs and ED, with a 38 percent increase in ED in patients who use NSAIDs on a regular basis (6). The authors warn that patients should not stop taking NSAIDs without consulting their physicians.

Also, high blood pressure drugs have a reputation for causing ED. A meta-analysis of 42 studies showed that beta blockers have a small effect, but thiazide diuretics (water pills) more than doubled ED, compared to placebo (7).

How can diet affect ED?

The Mediterranean-type diet has been shown to treat and prevent ED. It’s a green leafy alternative to the little blue pill. Foods are rich in omega-3 fatty acids and high in monounsaturated fats and polyunsaturated fats, as well as fiber. Components include whole grains, fruits, vegetables, legumes, walnuts, and olive oil. 

In two RCTs lasting two years, those who followed a Mediterranean-type diet experienced improvements in their endothelial functioning (8, 9). They also experienced both lower inflammation and lower insulin resistance.

In another study, those who had the highest compliance with a Mediterranean-type diet were significantly less likely to have ED, compared to those with the lowest compliance (10). Even more impressive was that the group with the highest compliance had a 37 percent reduction in severe ED versus the low-compliance group.

A study of participants in the Health Professionals Follow-up Study looked closely at both the Mediterranean-type diet and the Alternative Healthy Eating Index 2010 diet, which emphasized consuming vegetables, fruits, nuts, legumes, and fish or other sources of long-chain fats, as well as avoiding red and processed meats (11). At this point, it probably won’t surprise you to hear that the greater participants’ compliance with either of these diets, the less likely they were to experience ED.

References:

(1) clevelandclinic.org. (2) J Sex Med. 2024;21(4): 296–303. (3) J Sex Med. 2009;6(5):1414-22. (4) Int J Androl. 2010;33(6):853-60. (5) J Am Coll Cardiol. 2005;46(8):1503. (6) Medicine (Baltimore). 2018 Jul;97(28):e11367. (7) JAMA. 2002;288(3):351. (8) Int J Impot Res. 2006;18(4):405-10. (9) JAMA. 2004;292(12):1440-6. (10) J Sex Med. 2010 May;7(5):1911-7. (11) JAMA Netw Open. 2020 Nov 2;3(11):e2021701.

Dr. David Dunaief is a speaker, author and local lifestyle medicine physician focusing on the integration of medicine, nutrition, fitness and stress management. For further information, visit www.medicalcompassmd.com or consult your personal physician.

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Recently, towns across our coverage area have had to confront the loss innate in progress, especially as it affects the delicate balancing scale of Long Island’s development. In Smithtown, the Kings Park Revitalization Plan enters its final stages leaving some residents overjoyed and others worried about congestion.

Simultaneously, in Setauket, the controversial battery energy storage facilities are being opposed partly because they would be located in population dense areas. These events have triggered vastly differing reactions, but all raise questions: How much change is too much? And, is change worth the loss?

The heart of the opposition in each of these issues is that Long Island is under threat of overdevelopment, as it has been for years. Despite this, it is becoming unlivable due to financial strains. Ever present in discourse across every sector, is the exodus of young Long-Islanders–the skills we lose when each one moves away taking their education and expertise with them, the sorrow of having a loved one no longer within driving distance. 

The Kings Park Revitalization Plan struck the balance—nearly. Some people decry the development it promotes, while others say it is the very thing their hamlet needs and has the potential to bring life and energy back to their community, perhaps enticing young people to stay and build their lives, families and careers. 

Each viewpoint, especially the critical ones, smoothed the rough surfaces of the plan. Paying attention to defects is an essential step in ensuring that we don’t become so infatuated with progress that we forget what we have. 

Some people are still unhappy with the Kings Park Revitalization Plan as it enters its last step before it is voted on for approval. However, their voices help develop a more complete, well-informed opinion on what matters to our community. 

Public hearings may slow down the process, delaying approval, but they prompt analysis and re-analysis, reminding us of the trade offs we need to consider. 

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A Column Promoting a More Earth-Friendly Lifestyle

By John L. Turner

John Turner

If you’re like most homeowners much of the mail you receive is unsolicited and of no interest, what we typically refer to as junk mail. Not surprisingly, much of it is never opened or read (about 44% according to one website) and gets tossed — hopefully in the recycling bin. Americans receive,  and dispose of, about 3 million tons of junk mail every year. Especially notorious are catalogs offering various products  — gifts, clothing, etc. — since they use much more paper than what is contained in a mere solicitation letter offering you a new product you don’t want.   

Fortunately, there are several steps you can take to reduce the amount of unwanted mail you receive such as services which eliminate or reduce the amount of unwanted mail you receive. Some charge a small fee to process your request. One such service highlighted by the United States Postal Service is: DMAchoice, Consumer Preferences, P.O. Box 900, Cos Cob, CT 06807. 

If you want to opt out of unsolicited credit card and insurance company offers, call toll free 1-888-567-8688 or visit optoutprescreen.com. Also, if you don’t want Valpak coupons go to valpak.com/remove-address to unsubscribe. Lastly, download the phone app PaperKarma that helps you to unsubscribe from unwanted mail.

A resident of Setauket, author John L. Turner is a naturalist, conservation co-chair of the Four Harbors Audubon Society, and Conservation Policy Advocate for the Seatuck Environmental Association.

 

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By Bob Lipinski

Bob Lipinski

Yes, I know it’s winter, with bitter cold days and even colder nights, and some parts of the country are probably experiencing snow or ice, which makes things even colder and more miserable.

Now, do people near the equator only drink chilled white wine and those near the poles drink only room temperature red wine? Certainly not to both statements.

Yet, during cold weather, we still drink cold beer, and we serve most of our cocktails cold or very cold, especially a martini, which we drink “arctic cold.” So, what’s wrong with serving a glass of a chilled white or red wine?

Here are some chillable wines for cold weather…

2022 Brigid ‘Pinot Noir,’ Marlborough, New Zealand. (9.5% alcohol; 80 calories per serving) Has a “Nutrition Facts” chart, like on a box of cereal, displayed on the back label. Light ruby with notes of cranberry, dried flowers, citrus, and tart cherry with hints of herbs and wet earth. A flavorful, easy to drink wine. A great accompaniment to cheese fondue.

NV Riunite Lambrusco, Emilia-Romagna, Italy. A perennial favorite since the 1970s. Although I’m not a stranger to this wine, I’ve not had a bottle in many years. It’s frizzante (lightly effervescent) and semi-sweet, with a luscious aroma and flavor of raspberry, rhubarb, and plum, with a bitter almond aftertaste. Try with a dish of salami, mortadella and provolone.

2024 Georges Duboeuf ‘Beaujolais Nouveau,’ France. (Made from Gamay grapes). Bright red colored; intensely fruity with hints of cherry, cotton candy, hibiscus, and red lollipops. Aftertaste of boysenberry, red currants, and spices. Drink with a spicy pepper, grilled cheese sandwich.

2020 Domaine Sérol ‘Oudan’ Côte Roannaise, Loire Valley, France. (100% Gamay grapes.) Oudan is a plot of hillside land on granitic soil, first planted by Stéphane Sérol over 20 years ago. Cherry-colored with a fruity aroma and taste of strawberry, red apple, and herbs. Hints of black pepper, potting soil, and fennel. A Margherita pizza says it all!

2023 Quinta da Lixa, Alvarinho ‘Pouco Comum,’ Vinho Verde, DOC, Portugal. Perfumed, with notes of honeysuckle, white peach, lemon, orange, and pear. Hints of green peas and pineapple. Pairs well with fish tacos.

2023 Quinta d’Amares, ‘Loureiro’ Vinho Verde, DOC, Portugal. Light citrine color; notes of perfume, lemon, bay leaf, and apricot. Nuances of minerals, peach, and melon. A delightful wine for hot and cold weather. Serve with a hearty bowl of fish chowder.

Bob Lipinski is the author of 10 books, including “101: Everything You Need To Know About Whiskey” and “Italian Wine & Cheese Made Simple” (available on Amazon.com). He consults and conducts training seminars on Wine, Spirits, and Food and is available for speaking engagements. He can be reached at www.boblipinski.com OR [email protected].